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A CNIC study reveals that mutations previously identified in the gene MINDBOMB1 (MIB1) as causing non-compaction cardiomyopathy either provoke this disease or valve defects, depending on their combination with mutations in modifier genes

CNIC scientists have identified a molecular mechanism that accounts for the arrhythmias, skeletal muscle weakness, and periodic paralysis in patients with this rare disease

A new study defines a mechanism that regulates inflammation after a myocardial infarction, opening a path to the use of the receptor

A study, published in Nature Metabolism, is the first to demonstrate the key role played by mitochondria in adapting metabolism to different energy demands in humans

Metoprolol, an established drug in the cardiologists armory, protects the brain during a stroke and greatly reduces its consequences

CNIC's leader have been recognized for his exceptional career achievements at the 34^th annual conference

The findings, published in the Journal of the American College of Cardiology (JACC), will enable physicians to make appropriate treatment adjustments for patients with this disease